![]() ![]() ![]() When they combined the S-protein in vitro with neutrophil elastase-a proteolytic enzyme (protease) that is produced in the human body by a type of white blood cell released early in SARS-CoV-2 infection called neutrophils-the researchers found that the S-protein broke up into sections that could lead to amyloid production. They theorized that the S-protein would need to react with something for amyloids to form, such as an enzyme that would break down the S-protein into smaller components. The study authors note that the S-protein on its own would not likely break down into amyloids. The researchers found that several components of the S-protein could potentially produce amyloids. The spike protein or S-protein plays a major role in the virus’ transmission and evolution. In the present study, researchers sought to examine if the spike protein in SARS-CoV-2 could be converted to amyloids in the body. SARS-CoV-2 can cause a variety of symptoms in individuals and researchers do not fully understand why these symptoms develop or impact some people more than others. The findings suggest that some lingering COVID-19 symptoms may be related to amyloid production caused by the immune system interacting with the S-protein (spike protein) on the surface of the SARS-CoV-2 virus. This has made both the immune system and circulatory system interesting candidates for further study.Ī recent study, published in the Journal of American Chemical Society, has provided a suggested mechanism to explain why some people develop complicated COVID-19 symptoms after infection. Researchers have been looking into various systems in the body in an effort to find answers.Ī sometimes controversial area of study has been micro clots in people with long COVID, caused by fibrin, which is a substance that contributes to coagulation. The cause of the many mysterious and lingering symptoms brought on by SARS-CoV-2 infection, or COVID-19, has remained a hard-to-solve puzzle for scientists. Visit our coronavirus hub for the most recent information on the COVID-19 pandemic. All data and statistics are based on publicly available data at the time of publication. ![]()
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